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Myasthenia gravis (MG), a neuromuscular junction disorder, causes weakness in the voluntary muscles of the body involving eyes, mouth, throat, arms, and legs
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Myasthenia gravis (MG) is an autoimmune disease caused by antibodies directed at acetylcholine receptors or functionally related proteins in the postsynaptic membrane at the neuromuscular junction
1,2 The inability to achieve sufficient clinical improvement with minimal symptom expression
This can manifest in a number of ways and may be influenced by the antibody that is positive in the patient: ocular weakness: e
If you have MG, your immune system makes antibodies that block or change the connection between Myasthenia gravis (MG) is an autoimmune disease where antibodies against the postsynaptic membrane at the neuromuscular junction lead to muscle weakness [ 35 ]
Myasthenia gravis is one of the better understood neurological disorders, but it can be a Context and Policy Issues
Both may have similar clinical features
Being an autoimmune disease, MG correlates with the presence of detectable antibodies directed against the acetylcholine receptor, muscle-specific kinase, lipoprotein-related protein 4, agrin, titin, and ryanodine Myasthenia gravis (MG) is a relatively rare autoimmune disorder in which antibodies form against acetylcholine nicotinic postsynaptic receptors at the neuromuscular junction of skeletal muscles (see the image below)
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Myasthenia gravis (MG) is an autoimmune neuromuscular disorder characterized by fluctuating motor weakness involving ocular, bulbar, limb, and/or respiratory muscles